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The decision to build a heavy-ion accelerator in Japan dedicated to the treatment of cancer has been controversial: The facility was costly to build, and the therapy is still experimental.
The recent descriptions of T-cell dynamics in HIV disease have refocused efforts to understand the normal homeostatic processes that maintain T-cell populations (pages 674–680).
Understanding the genetic defects underlying cystic fibrosis is only half the battle. Identifying the specific bacterium infecting CF patients is just as important (pages 661–666).
The promising description of a potential basis for gene therapy in treating HIV infection does not mean that traditional approaches should be abandoned (pages 667–673).
The gains made in life expectancy and against infant mortality in low-income nations are being accompanied by increases in mental-health-related problems.
Mutations in tumour suppressors often lead to tumorigenesis. But other genetic mechanisms affecting suppressor gene expression can be just as effective (pages 686–692).
The role of the CD44 cell surface molecule in tumorigenesis has been the focus of intense debate. Now enough pieces are known to begin putting the puzzle together.
