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Fibroblasts from patients with schizophrenia have been reprogrammed into neurons, and reveal expected and unexpected changes in neuronal connectivity and gene expression.
New evidence indicates that mutations in the postsynaptic density protein SHANK3 causes defects in corticostriatal signalling and autism-like behaviours in mice.
Murine β-adducin, and its fly homologue HTS, have key roles in synapse assembly and disassembly, and mediate the improvement of learning and memory induced by environmental enrichment.
A neural correlate between aggressive behaviour and mating has been found. This involves a set of neurons in the ventromedial hypothalamus that are active during fighting and suppressed during mating.