Mechanisms and actions of ferroptosis
- Submission status
- Open
- Submission deadline
Discovered over a decade ago, ferroptosis is a form of non-apoptotic cell death that depends on the production of reactive oxygen species (ROS), iron and lipid peroxidation. Ferroptosis has been a very attractive research target ever since as it has a central position in cellular (lipid) metabolism and oxidative stress homeostasis and employs several cellular organelles from the endoplasmic reticulum (ER), the Golgi, peroxisomes and lysosomes to mitochondria. Importantly, ferroptosis is implicated in a variety of diseases and pathophysiological conditions of the nervous and cerebrovascular system, kidney repair and cancer, with an attractive avenue for anti-tumour therapy.
For this Collection, we invite submissions on cellular signalling and mechanisms of ferroptosis and its roles in a pathological context. In addition to original research Articles, we will also consider Reviews, Perspectives and Comments covering these topics. All submissions will be subject to the same review process as regular Communications Biology Articles.
This Collection supports and amplifies research related to SDG 3.
The Collection will publish original research Articles, Reviews, Perspectives and Comments (full details on content types can be found here). Papers will be published in Communications Biology as soon as they are accepted and then collected together and promoted on the Collection homepage. Collections are associated with a call for papers and are managed by one or more of our Editorial Board Members and the journal's Editors.
This Collection welcomes submissions from all authors – and not by invitation only – on the condition that the manuscripts fall within the scope of the Collection and of Communications Biology more generally. See our editorial process page for more details.
All submissions are subject to the same peer review process and editorial standards as regular Communications Biology articles, including the journal’s policy on competing interests. See our Collections guidelines for more details.
This Collection is not supported by sponsorship.
