An iron-dependent form of cell death called ferroptosis has been implicated as a component of the tumour-suppressor activity of p53, providing fresh insight into how this protein prevents cancer development. See Article p.57
This is a preview of subscription content, access via your institution
Relevant articles
Open Access articles citing this article.
-
The Pu.1 target gene Zbtb11 regulates neutrophil development through its integrase-like HHCC zinc finger
Nature Communications Open Access 06 April 2017
Access options
Subscribe to this journal
Receive 51 print issues and online access
$199.00 per year
only $3.90 per issue
Buy this article
- Purchase on Springer Link
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Notes
References
Freed-Pastor, W. A. & Prives, C. Genes Dev. 26, 1268–1286 (2012).
Jiang, L. et al. Nature 520, 57–62 (2015).
Lane, D. P. Nature 358, 15–16 (1992).
Vousden, K. H. & Prives, C. Cell 137, 413–431 (2009).
Brady, C. A. et al. Cell 145, 571–583 (2011).
Li, T. et al. Cell 149, 1269–1283 (2012).
Valente, L. J. et al. Cell Rep. 3, 1339–1345 (2013).
Conrad, M. & Sato, H. Amino Acids 42, 231–246 (2012).
Dixon, S. J. et al. Cell 149, 1060–1072 (2012).
Bieging, K. T., Mello, S. S. & Attardi, L. D. Nature Rev. Cancer 14, 359–370 (2014).
Yang, W. S. et al. Cell 156, 317–331 (2014).
Author information
Authors and Affiliations
Corresponding author
Rights and permissions
About this article
Cite this article
Bieging, K., Attardi, L. A piece of the p53 puzzle. Nature 520, 37–38 (2015). https://doi.org/10.1038/nature14374
Published:
Issue Date:
DOI: https://doi.org/10.1038/nature14374
This article is cited by
-
The Pu.1 target gene Zbtb11 regulates neutrophil development through its integrase-like HHCC zinc finger
Nature Communications (2017)