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This study suggests that brain-derived neurotrophic factor (BDNF) is released from dendritic spines in response to activity and acts in an autocrine manner to mediate structural plasticity of the spine from which it was released.
A loss of cholinergic inputs to the hippocampus in a mouse model of Alzheimer disease leads to memory deficits by affecting interneurons in the oriens lacunosum moleculare.
Sean Hill explains why the growing importance of data-intensive neuroscience makes it crucial for the community to establish minimal metadata standards
Inputs to the ventral tegmental area, which computes the reward prediction error, carry diverse signals about the actual and expected reward from distributed areas.
Early in development, GABA released from interneurons is excitatory and induces the formation of functional inhibitory and excitatory synapses in cortical layer 2/3.
Adult neurogenesis in the hippocampus is regulated by existing hippocampal connectivity and can be manipulated to enhance memory function in aged mice.