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Analyses of prion protein deposits in patients with Creutzfeldt-Jakob disease contribute to the controversy over the nature of the transmissible agent (pages 521–525).
New evidence implicates salt-sensitive, natural antibiotics in lung mucosal defenses and may explain how a defective chloride channel leads to lung infections.
Implants of polymer-encapsulated cells producing CNTF protect neurons of the striatum and cortex from drug-induced destruction in a monkey model of Huntington's disease.
