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Volume 7 Issue 5, May 2006

The continued spread of the H5N1 influenza virus emphasizes the importance of anti-influenza immune defense mechanisms. Three papers in this month's issue of Nature Immunology examine the immune cell response to influenza virus infection and discuss the challenge this virus poses to population-based immunity (pp 443, 449 and 517). Image shows an agglomeration of influenza viruses. Artwork by Lewis Long.

Volume 7 Issue 5

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  • Contact hypersensitivity is a form of delayed-type hypersensitivity, a classic T cell–mediated, clinically important phenomenon. Unexpectedly, a new study indicates that natural killer cells may mediate contact hypersensitivity and demonstrate adaptive, memory-like activity.

    • Wayne M Yokoyama
    News & Views

  • The resolution of immune responses typically leaves a population of memory T cells to respond to subsequent infection. The generation of 'memory-like' T cells can also occur during homeostatic proliferation, but are they 'true' memory cells?

    • Charles D Surh
    • Jonathan Sprent
    News & Views

  • Plasmacytoid dendritic cells selectively express Toll-like receptors 7 and 9 and respond to virus infection by triggering massive interferon production, a pathway regulated by osteopontin expressed in plasmacytoid dendritic cells.

    • Wei Cao
    • Yong-Jun Liu
    News & Views

  • B lymphocyte–induced maturation protein 1 (Blimp-1) is known as a 'master regulator' of plasma cell differentiation. New findings suggest that the influence of this transcription factor extends beyond the B cell lineage.

    • Pamela J Fink
    News & Views
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