Featured Articles

Receptors involved in attention strengthen working memory by closing ion channels in dendritic spines.

Excess neurogenesis at the cost of gliogenesis may cause cognitive dysfunction in Noonan syndrome.

Neurons that contain the most CREB have a competitive advantage over neighboring neurons in memory formation.

Mutations that reduce cAMP-dependent protein kinase activity suppress memory loss in middle-aged Drosophila.

A sex pheromone specifically activates one type of olfactory receptor neuron in Drosophila that inhibits courtship behavior in males but promotes it in females.

Inhibiting GABA-mediated inhibition improves learning and memory in a mouse model of Down syndrome.

Mutations that impair learning also affect selective attention in Drosophila.

Epigenetic modification of DNA is important in memory formation.

A drug that disrupts the association of androgen receptor with its cofactors improves motor behavior in a mouse model of spinal and bulbar muscular atrophy.

Forward genetic screening identifies recessive mutations associated with deafness.

Data from over 1400 families identify genetic regions associated with autism spectrum disorders.

The immune molecule CD38 is important in social behaviors because it regulates the release of oxytocin.

Researchers rescue the neurological phenotype in a mouse model of Rett syndrome.

Genes associated with basolateral amygdala morphology are involved in neuronal development.

Variable levels of Notch signaling define the gene expression and synaptic targets of olfactory receptor neurons.

Alleles of a gene involved in recycling amyloid precursor protein from the cell surface associate with familial late-onset Alzheimer disease.

High-throughput behavioral screening of mouse mutants identifies a mutation in α-tubulin that may cause a neurodevelopmental disorder in humans.

Drosophila detect carbon dioxide with two receptors from the taste receptor family of genes.

Variations in pain sensitivity associate with different secondary structures of an enzyme mRNA.

Different mutations in the same sodium channel gene may cause excessive pain or the inability to sense pain.