Liver regeneration can occur by proliferation of two different cell types, but generally only one at a time is fully turned on. A factor that may specifically activate one of these cell types now comes to light.
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 12 print issues and online access
$209.00 per year
only $17.42 per issue
Buy this article
- Purchase on Springer Link
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Katie Ris
References
Jakubowski, A. et al. J. Clin. Invest. 115, 2330–2340 (2005).
Fausto, N. Hepatology 39, 1477–1487 (2004).
Knight, B. and Yeoh, G.C. Cell Tissue Res. 319, 61–70 (2005).
Feng, S.L. et al. Am. J. Pathol. 156, 1253–1261 (2000).
Akhurst, B. et al. Hepatology 41, 327–335 (2005).
Anders, R.A. et al. J. Immunol. 175, 1295–1300 (2005).
Brooling, J.T. et al. Hepatology 41, 906–915 (2005).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Fausto, N. Tweaking liver progenitor cells. Nat Med 11, 1053–1054 (2005). https://doi.org/10.1038/nm1005-1053
Issue Date:
DOI: https://doi.org/10.1038/nm1005-1053
This article is cited by
-
Reduced circulating levels of sTWEAK are associated with NAFLD and may affect hepatocyte triglyceride accumulation
International Journal of Obesity (2016)
-
Inflammation and liver tumorigenesis
Frontiers of Medicine (2013)
-
TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration
The EMBO Journal (2006)
-
Current status and perspective of antiangiogenic therapy for cancer: hepatocellular carcinoma
International Journal of Clinical Oncology (2006)
