This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 12 print issues and online access
$209.00 per year
only $17.42 per issue
Buy this article
- Purchase on Springer Link
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
References
Wahl, A.F. et al. Loss of normal p53 function confers sensitization to Taxol by increasing G2/M arrest and apoptosis. Nature Med. 2, 72–79 (1996).
Hawkins, D.S., Demers, G.W. & Galloway, D.A. Inactivation of p53 enhances sensitivity to multiple chemotherapeutic agents. Cancer Res. 56, 892–898 (1996).
Wu, S.G. & El-Deiry, W.S. p53 and chemosensitivity. Nature Med. 2, 255–256 (1996).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Delia, D., Mizutani, S., Lamorte, G. et al. p53 activity and chemotherapy. Nat Med 2, 724–725 (1996). https://doi.org/10.1038/nm0796-724
Published:
Issue Date:
DOI: https://doi.org/10.1038/nm0796-724
This article is cited by
-
p53 acetylation enhances Taxol-induced apoptosis in human cancer cells
Apoptosis (2013)
-
A serine 37 mutation associated with two missense mutations at highly conserved regions of p53 affect pro-apoptotic genes expression in a T-lymphoblastoid drug resistant cell line
Oncogene (2000)
-
Reply to “Cell status — dead or alive?”
Nature Medicine (1996)
-
Cell status — dead or alive?
Nature Medicine (1996)