Abstract
The 'endothelial' isoform of nitric oxide synthase modulates (but is not obligatory for) cardiac muscle function; signaling by NO in different cellular compartments coordinates diverse effects within the cardiac myocyte (pages 331–334).
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Hare, J., Stamler, J. NOS: Modulator, not mediator of cardiac performance. Nat Med 5, 273–274 (1999). https://doi.org/10.1038/6486
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DOI: https://doi.org/10.1038/6486
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