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Upregulation of metabolic stress genes could underlie the dysregulation in stem cell differentiation and maturation observed in organoids.

In mice, inputs to the hippocampal CA1 region from the locus coeruleus have a key role in reorganizing place cell responses during spatial reward learning.

In a mouse model of prion disease, blocking a major branch of the unfolded protein response selectively in astrocytes was highly neuroprotective, reducing neurodegeneration, reducing astrocyte reactivity and increasing longevity.

One of two anatomically and functionally characterized subpopulations of neurons in the mouse paraventricular thalamus forms a thalamo-corticothalamic loop with the infralimbic cortex that regulates arousal.

Fear extinction memories are formed and stored within a population of reward-encoding neurons in the basolateral amygdala.

In mice, temporally patterned inputs to the motor cortex that controls the forelimb are required for a well-trained reaching behaviour.