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Epigenetic mechanisms do not control viral latency III in primary effusion lymphoma cells infected with a recombinant Epstein–Barr virus

Leukemia volume 19, pages 1854–1856 (2005)Cite this article

TO THE EDITOR

Epstein–Barr virus (EBV) is mainly associated with Burkitt lymphoma (BL), nasopharyngeal carcinoma (NPC), Hodgkin's disease (HD).1 This virus is the cause of lymphoproliferations arising in immunocompromised individuals. It is also strongly associated with diffuse large cell lymphomas (DLCL) and primary effusion lymphomas (PEL).2

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Acknowledgements

This work was supported by grants from the MIUR, Ministero della Sanità, Progetto AIDS, Associazione Italiana di ricerca sul Cancro (AIRC) and Istituto-Cenci-Bolognetti foundation. We gratefully acknowledge Drs Kenzo Takada and Antonino Carbone for providing the recombinant EBV strain and PEL cell lines, respectively. Miss Claudia Zompetta and Mr Sandro Valia provided skillful technical assistance.

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Authors and Affiliations

  1. Department of Experimental Medicine and Pathology, University of Rome, ‘La Sapienza’, Rome, Italy

    E Anastasiadou, F Boccellato, M Cirone, L Frati, A Faggioni & P Trivedi

  2. Microbiology and Tumor Biology center (MTC), Karolinska Institute, Stockholm, Sweden

    L L Kis & E Klein

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  1. E Anastasiadou
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  2. F Boccellato
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Correspondence to P Trivedi.

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Anastasiadou, E., Boccellato, F., Cirone, M. et al. Epigenetic mechanisms do not control viral latency III in primary effusion lymphoma cells infected with a recombinant Epstein–Barr virus. Leukemia 19, 1854–1856 (2005). https://doi.org/10.1038/sj.leu.2403895

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