Abstract
Hypoglycemia (H) in birth asphyxiated infants is commonly attributed to glycogen depletion. We have observed 3 term AGA infants with birth asphyxia, who developed hyperinsulinemic hypoglycemia (HH) in the early postnatal life. All had inappropriatelly high serum insulin concentrations for their blood glucose level, and needed glucose infusion rates of>8 mg/kg/min for several days to maintain normoglycemia. All infants recovered spontaneously.
It is important to consider HH in asphyxiated new-borns and vigorous treatment of H is needed to prevent CNS damage.
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Schultz, K., Soltész, G. TRANSIENT HYPERINSULINISM IN ASPHYXIATED NEWBORN INFANTS. Pediatr Res 22, 241 (1987). https://doi.org/10.1203/00006450-198708000-00170
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DOI: https://doi.org/10.1203/00006450-198708000-00170