Abstract
Summary: Fetal rat kidney showed glycogen deposition that reached a maximal value of 60 μ g·mg prot−1 on day 18 and declined thereafter. At birth glycogen concentration is reduced (20 μ g·mg prot−1) but higher than adult one (cortex, 2.2 μ g·mg prot−1 and medulla, 3.4 μ g·mg prot−1). From day 17 to the birth, glycogen synthetase and phosphorylase activities increased slowly except for acid glucosidase activity which increased rapidly between day 18 to the birth (3-fold). Corticosteroid deprivation had no effect upon glycogen content but fetal decapitation on day 16 reduced glycogen content in kidney of 19-day-old fetuses.
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Delaval, E., Moreau, E., Andriamanantsara, S. et al. Renal Glycogen Content and Hormonal Control of Enzymes Involved in Renal Glycogen Metabolism. Pediatr Res 17, 766–769 (1983). https://doi.org/10.1203/00006450-198309000-00017
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DOI: https://doi.org/10.1203/00006450-198309000-00017
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