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Inflammatory mechanisms in Alzheimer disease: the role of beta-amyloid/glial interactions

Molecular Psychiatry volume 2, pages 91–95 (1997)Cite this article

Abstract

Recent evidence that insoluble aggregates of beta-amyloid peptide in the brains of Alzheimer patients may elicit neurodestructive immune responses offers the prospect of new therapies to treat this neurodegenerative condition.

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  1. Department of Pharmacology, University of Connecticut Health Center, 263 Farmington Ave, Farmington, 06030, CT, USA

    J S Pachter

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  1. J S Pachter
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Pachter, J. Inflammatory mechanisms in Alzheimer disease: the role of beta-amyloid/glial interactions. Mol Psychiatry 2, 91–95 (1997). https://doi.org/10.1038/sj.mp.4000212

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