Abstract
IT is known that adrenaline accelerates glycogenolysis in liver and in muscles, which leads to an elevation of the concentration of blood sugar and blood lactate1. During work (and especially with high work loads) there is a substantial increase in the concentration of blood lactate, which is mainly produced in the working muscles2. This can in part arise from increased adrenergic activity during work. The purpose of the present investigation was to test this assumption by means of an adrenergic beta-blocking agent.
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Goodman, L. S., and Gilman, A., The Pharmacological Basis of Therapeutics (The Macmillan Company of New York, 1958).
Best, C. H., and Taylor, N. B., The Physiological Basis of Medical Practice (The Williams and Wilkins Company, Baltimore, 1955).
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FURBERG, C. Adrenergic Beta-receptor Blockade and Anaerobic Metabolism. Nature 211, 888 (1966). https://doi.org/10.1038/211888a0
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DOI: https://doi.org/10.1038/211888a0
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