Volume 44 Issue 7, July 2023
Cover Credit: Defect in branched-chain amino acid (BCAA) catabolism has been recognized as a metabolic hallmark and therapeutic target for heart failure. As BCAA catabolic enzymes express ubiquitously, it is important to determine the cell-autonomous impact of BCAA catabolic defect in cardiomyocytes in vivo. In this issue, Chen Gao and the colleagues explored the cardiomyocytes specific BCAA catabolic defects in vivo.
