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Research | 28 January 2019
An allosteric MALT1 inhibitor is a molecular corrector rescuing function in an immunodeficient patient
- Jean Quancard
- , Theo Klein
- , Shan-Yu Fung
- , Martin Renatus
- , Nicola Hughes
- , Laura Israël
- , John J. Priatel
- , Sohyeong Kang
- , Michael A. Blank
- , Rosa I. Viner
- , Jutta Blank
- , Achim Schlapbach
- , Paul Erbel
- , Jayachandran Kizhakkedathu
- , Frédéric Villard
- , René Hersperger
- , Stuart E. Turvey
- , Joerg Eder
- , Frédéric Bornancin
- & Christopher M. Overall
Nature Chemical Biology 15 , 304–313

Rights & permissionsfor article An allosteric MALT1 inhibitor is a molecular corrector rescuing function in an immunodeficient patient . Opens in a new window.
Research | 20 June 2018 | Open
C-terminal truncation of IFN-γ inhibits proinflammatory macrophage responses and is deficient in autoimmune disease
- Antoine Dufour
- , Caroline L. Bellac
- , Ulrich Eckhard
- , Nestor Solis
- , Theo Klein
- , Reinhild Kappelhoff
- , Nikolaus Fortelny
- , Parker Jobin
- , Jacob Rozmus
- , Jennifer Mark
- , Paul Pavlidis
- , Vincent Dive
- , Sean J. Barbour
- & Christopher M. Overall
Nature Communications 9 , 1–18

Rights & permissionsfor article C-terminal truncation of IFN-γ inhibits proinflammatory macrophage responses and is deficient in autoimmune disease . Opens in a new window.
Research | 26 June 2018 | Open
Granzyme B is elevated in autoimmune blistering diseases and cleaves key anchoring proteins of the dermal-epidermal junction
- Valerio Russo
- , Theo Klein
- , Darielle J. Lim
- , Nestor Solis
- , Yoan Machado
- , Sho Hiroyasu
- , Layla Nabai
- , Yue Shen
- , Matthew R. Zeglinski
- , Hongyan Zhao
- , Cameron P. Oram
- , Peter A. Lennox
- , Nancy Van Laeken
- , Nick J. Carr
- , Richard I. Crawford
- , Claus-Werner Franzke
- , Christopher M. Overall
- & David J. Granville
Scientific Reports 8 , 1–11

Rights & permissionsfor article Granzyme B is elevated in autoimmune blistering diseases and cleaves key anchoring proteins of the dermal-epidermal junction . Opens in a new window.
Research | 3 November 2015 | Open
The paracaspase MALT1 cleaves HOIL1 reducing linear ubiquitination by LUBAC to dampen lymphocyte NF-κB signalling
- Theo Klein
- , Shan-Yu Fung
- , Florian Renner
- , Michael A. Blank
- , Antoine Dufour
- , Sohyeong Kang
- , Madison Bolger-Munro
- , Joshua M. Scurll
- , John J. Priatel
- , Patrick Schweigler
- , Samu Melkko
- , Michael R. Gold
- , Rosa I. Viner
- , Catherine H. Régnier
- , Stuart E. Turvey
- & Christopher M. Overall
Nature Communications 6 , 1–17

Rights & permissionsfor article The paracaspase MALT1 cleaves HOIL1 reducing linear ubiquitination by LUBAC to dampen lymphocyte NF-κB signalling . Opens in a new window.

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An allosteric MALT1 inhibitor is a molecular corrector rescuing function in an immunodeficient patient

C-terminal truncation of IFN-γ inhibits proinflammatory macrophage responses and is deficient in autoimmune disease

Granzyme B is elevated in autoimmune blistering diseases and cleaves key anchoring proteins of the dermal-epidermal junction

The paracaspase MALT1 cleaves HOIL1 reducing linear ubiquitination by LUBAC to dampen lymphocyte NF-κB signalling

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