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Showing 1–12 of 12 results

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Reviews | 1 February 2010
T-cell lymphoproliferative disorder potentially induced by imatinib in a patient with GIST
- justus.duyster@lrz.tum.de
Nature Reviews Clinical Oncology 7 , 116–119

Rights & permissionsfor article T-cell lymphoproliferative disorder potentially induced by imatinib in a patient with GIST . Opens in a new window.
Research | 24 July 2003
The oncogenic fusion protein nucleophosmin–anaplastic lymphoma kinase (NPM–ALK) induces two distinct malignant phenotypes in a murine retroviral transplantation model
- justus.duyster@lrz.tum.de
Oncogene 22 , 4642–4647

Rights & permissionsfor article The oncogenic fusion protein nucleophosmin–anaplastic lymphoma kinase (NPM–ALK) induces two distinct malignant phenotypes in a murine retroviral transplantation model . Opens in a new window.
Research | 11 February 2002
SMIF, a Smad4-interacting protein that functions as a co-activator in TGFβ signalling
- justus.duyster@lrz.tum.de
Nature Cell Biology 4 , 181–190

Rights & permissionsfor article SMIF, a Smad4-interacting protein that functions as a co-activator in TGFβ signalling . Opens in a new window.
Research | 27 June 2002
Analysing c-kit internalization using a functional c-kit-EGFP chimera containing the fluorochrome within the extracellular domain
- justus.duyster@lrz.tum.de
- Thomas Jahn
- , Petra Seipel
- , Sunita Coutinho
- , Susanne Urschel
- , Kathleen Schwarz
- , Cornelius Miething
- , Hubert Serve
- , Christian Peschel
- & Justus Duyster
Oncogene 21 , 4508–4520

Rights & permissionsfor article Analysing c-kit internalization using a functional c-kit-EGFP chimera containing the fluorochrome within the extracellular domain . Opens in a new window.
Reviews | 10 September 2001
Translocations involving anaplastic lymphoma kinase (ALK)
- justus.duyster@lrz.tum.de
Oncogene 20 , 5623–5637

Rights & permissionsfor article Translocations involving anaplastic lymphoma kinase (ALK) . Opens in a new window.
Research | 5 April 2016
Lineage-specific STAT5 target gene activation in hematopoietic progenitor cells predicts the FLT3⁺-mediated leukemic phenotype
- Justus.Duyster@uniklinik-freiburg.de
- T A Müller
- , R Grundler
- , R Istvanffy
- , M Rudelius
- , L Hennighausen
- , A L Illert
- & J Duyster
Leukemia 30 , 1725–1733

Rights & permissionsfor article Lineage-specific STAT5 target gene activation in hematopoietic progenitor cells predicts the FLT3<sup>+</sup>-mediated leukemic phenotype . Opens in a new window.
Correspondence | 16 December 2004
Myeloid blast crisis evolving during imatinib treatment of an FIP1L1-PDGFR alpha-positive chronic myeloproliferative disease with prominent eosinophilia
- justus.duyster@lrz.tu-muenchen.de
- N von Bubnoff
- , M Sandherr
- , G Schlimok
- , R Andreesen
- , C Peschel
- & J Duyster
Leukemia 19 , 286–287

Rights & permissionsfor article Myeloid blast crisis evolving during imatinib treatment of an FIP1L1-PDGFR alpha-positive chronic myeloproliferative disease with prominent eosinophilia . Opens in a new window.
Correspondence | 16 January 2013
The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD⁺ AML but retains in vitro sensitivity to PKC412 and Sunitinib
- justus.duyster@lrz.tum.de
- C Albers
- , H Leischner
- , M Verbeek
- , C Yu
- , A L Illert
- , C Peschel
- , N von Bubnoff
- & J Duyster
Leukemia 27 , 1416–1418

Rights & permissionsfor article The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD<sup>+</sup> AML but retains <i>in vitro</i> sensitivity to PKC412 and Sunitinib . Opens in a new window.
Correspondence | 30 June 2005
Combination of imatinib with rapamycin or RAD001 acts synergistically only in Bcr-Abl-positive cells with moderate resistance to imatinib
- justus.duyster@lrz.tum.de
- J Dengler
- , N von Bubnoff
- , T Decker
- , C Peschel
- & J Duyster
Leukemia 19 , 1835–1838

Rights & permissionsfor article Combination of imatinib with rapamycin or RAD001 acts synergistically only in Bcr-Abl-positive cells with moderate resistance to imatinib . Opens in a new window.
Correspondence | 14 July 2005
The systemic mastocytosis-specific activating cKit mutation D816V can be inhibited by the tyrosine kinase inhibitor AMN107
- justus.duyster@lrz.tum.de
- N von Bubnoff
- , S H P Gorantla
- , R K Kancha
- , F Lordick
- , C Peschel
- & J Duyster
Leukemia 19 , 1670–1671

Rights & permissionsfor article The systemic mastocytosis-specific activating cKit mutation D816V can be inhibited by the tyrosine kinase inhibitor AMN107 . Opens in a new window.
Research | 16 February 2006
The Bcr-Abl mutations T315I and Y253H do not confer a growth advantage in the absence of imatinib
- justus.duyster@lrz.tum.de
- C Miething
- , S Feihl
- , C Mugler
- , R Grundler
- , N von Bubnoff
- , F Lordick
- , C Peschel
- & J Duyster
Leukemia 20 , 650–657

Rights & permissionsfor article The Bcr-Abl mutations T315I and Y253H do not confer a growth advantage in the absence of imatinib . Opens in a new window.
Comments & Opinion | 30 August 2002
Effects of imatinib on bone marrow engraftment in syngeneic mice
- justus.duyster@LRZ.TUU.DE
- J Hoepfl
- , C Miething
- , R Grundler
- , KS Götze
- , C Peschel
- & J Duyster
Leukemia 16 , 1584–1588

Rights & permissionsfor article Effects of imatinib on bone marrow engraftment in syngeneic mice . Opens in a new window.

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