In Brief

This study finds that toxin–antitoxin systems can protect bacteria from phages by inducing abortive infection.

A study with honey bee larvae shows that a bacterial symbiont can protect them from nutritional stress.

A microfluidics study shows that bacterial biofilms form wrinkles when they grow and detach from a solid surface.

A single-cell microfluidics study shows that phenotypic heterogeneity and fast growth can reduce antibiotic susceptibility.

A study in hamsters shows prolonged inflammatory changes after COVID-19.

A study in mice links a metabolite of the gut microbiota to age-related microglia death.

A study in mice finds that antibiotic treatment predisposes to infection with bacteria that are associated with hospital-acquired infection.

A retrospective analysis of seven cases of monkeypox describes virus loads over time and looks at potential management options.

An analysis of the economic impact of biofilms estimates it at almost US$4,000 billion per year globally.

A study looking at the Tibetan Plateau finds that microbial shifts might contribute to irreversible soil degradation.

This study used engineered bacteria to record the transcriptional responses of transiting bacteria in the gut environment.

A study in mice shows that antibiotic treatment predisposes to invasive fungal infection through gut dysbiosis and barrier dysfunction.

Experimental evolution experiments show that antibiotic resistance development decreases thermal tolerance and niche breadth.

A study in germ-free mice shows that the gut microbiota can influence dietary choices of the host.

An in vitro study shows that the nuclear lamina regulates Epstein–Barr virus latency and viral gene expression.

This study reports the discovery of two new DNA defence modules, termed DdmABC and DdmDE, in Vibrio cholerae.

This study suggests that fungicide treatment of stored fruit could contribute to the selection of resistant Candida auris.

This study uncovers how Yersinia pestis is protected from engulfment by macrophages by the capsule-like antigen F1.

A strain-level study shows transfer of antibiotic-resistance genes between gut commensals and pathogens.

A CRISPR–Cas screen identifies a novel C. difficile toxin receptor.