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In this article series, Nature Reviews Molecular Cell Biology looks at the progress that has been made in identifying resident stem cells in different tissues and in characterizing these often-heterogeneous stem cell pools, as well as in understanding the intrinsic mechanisms and interactions with stem cell niches (the specialized microenvironments in which they reside) that regulate stem cell function.
NRMCB is 20! To mark this anniversary, we selected Review articles in 10 core areas of the journal. The Reviews are presented in pairs, an older and a more recent one, alongside 10 commissioned Journal Clubs — together the articles showcase developments and key discoveries in each research field.
A series of short articles that provides a platform for junior group leaders to share their experiences and thoughts on their careers, and to offer advice on how to navigate in a challenging and rapidly-evolving research environment. Come back to this page as we add more content!
A series of Reviews and Comments on the molecular and cellular changes that occur during ageing and ageing-associated disorders, and on how targeting these changes could modulate healthspan and lifespan.
Metabolic pathways are dynamically regulated to meet the energy needs of the cell and maintain cell and organismal homeostasis. This series features Review articles that discuss various aspects of the regulation of metabolic signalling, the interface between metabolic regulators and cellular processes and the implications of their deregulation for human diseases.
The articles in this series discuss new insights that have been gained into how ‘old and new’ modifications are regulated and recognized, and how they crosstalk with one another to control fundamental biological processes.
In this article series, Nature Reviews Molecular Cell Biology explores the pathways that preserve genomic integrity by detecting and repairing damage in different types of cellular DNA, highlighting new control mechanisms in the DNA damage response and the implications of disrupted repair pathways for disease.