A postdoctoral position is available to study the pathogenesis of Borrelia burgdorferi, the causative agent of Lyme disease. Specifically, the project will focus on the cellular mechanisms associated with macrophage phagosomal signaling during infection with B. burgdorferi and other pathogenic spirochetes (e.g. Treponema pallidum). Additionally, studies will also concentrate on the associated innate signaling mechanisms in the murine infection model of Lyme disease. The research will be performed in the Department of Pediatrics at the UConn Health, Farmington, CT. The project will utilize mouse models, murine and human macrophages, and various cell culture-based techniques to investigate innate signaling pathways that contribute to the pathogenesis of Lyme disease. A Ph.D., as well as a strong background in cell culture, microscopy, microbiology, molecular biology, and genomic analysis is required. A background in innate immunity is desirable.
Please email curriculum vitae and the names, telephone numbers, and e-mail addresses of references to Dr. Juan C. Salazar (firstname.lastname@example.org), UConn Health, Farmington, CT.
UConn Health is an affirmative action employer, in addition to an EEO and M/F/V/PWD/PV employer.