Scientific Reports 7: Article number: 46667; published online: 20 April 2017; updated: 22 December 2017.

The original version of this Article contained errors in the numbering of affiliations where Affiliations 1 and 2 were reversed.

In addition, the title and legend of Figure 4 contained errors.

Figure 4: IL-8 is reduced in human small airway epithelial cells(HSAEpC) from COPD patients. (a) The IL-8 protein levels were reduced in human small airway epithelial cells derived from COPD(COPD HSAEpCs) patients compared with normal HSAEpCs(NC). The levels of IL-8 in supernatants were measured by ELISA. (b) However, the IL-10 levels in COPD HSAEpCs were similar to in normal control cells. (c) The recruitment of SUV39H1 in the IL-8 gene promoter was reduced in COPD HSAEpCs compared with normal control. The enrichment of SUV39H1 was evaluated by chromatin immunoprecipitation(ChIP) analysis. (d) The enrichment of H3K9me3 in IL-8 gene promoter was also reduced in COPD HSAEpCs. All data are presented as the mean ± SEM of at least three independent experiments. **p < 0.01 and ***p < 0.001”.

now reads:

Figure 4: IL-8 is induced in human small airway epithelial cells(HSAEpC) from COPD patients. (a) The IL-8 protein levels were induced in human small airway epithelial cells derived from COPD(COPD HSAEpCs) patients compared with normal HSAEpCs(NC). The levels of IL-8 in supernatants were measured by ELISA. (b) However, the IL-10 levels in COPD HSAEpCs were similar to in normal control cells. (c) The recruitment of SUV39H1 in the IL-8 gene promoter was reduced in COPD HSAEpCs compared with normal control. The enrichment of SUV39H1 was evaluated by chromatin immunoprecipitation(ChIP) analysis. (d) The enrichment of H3K9me3 in IL-8 gene promoter was also reduced in COPD HSAEpCs. All data are presented as the mean ± SEM of at least three independent experiments. **p < 0.01 and ***p < 0.001”.

These errors have now been corrected in the HTML and PDF versions of the Article.