Correction to: Communications Biology https://doi.org/10.1038/s42003-020-01538-4, published online 8 January 2021.
The original version of this Article contained an error in the variant mentioned in the following text from the “Introduction”: “Notably, a similar Mg2+ block attenuating point mutation (c.1841A>G, p.Asn615Ser) at the identical position of the GluN2A subunit was found in two unrelated young female patients who suffered from epileptic seizures...”. The correct variant is (c.1845C>A, p.Asn615Lys).
The original version of this Article also contained an error in the mutation mentioned in the following text from the “Results” section “Generation of GluNA (N615S)-expressing mice”: “By classical gene-targeted replacement28 we inserted the c.1841A>G mutation at the homologous position in exon 10…”. The correct position of this mutation is c.1844A>G.
Both errors have been corrected in the HTML and PDF versions of the Article.
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Bertocchi, I., Eltokhi, A., Rozov, A. et al. Author Correction: Voltage-independent GluN2A-type NMDA receptor Ca2+ signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Commun Biol 4, 187 (2021). https://doi.org/10.1038/s42003-021-01717-x
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DOI: https://doi.org/10.1038/s42003-021-01717-x
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