Correction to: Scientific Reports https://doi.org/10.1038/srep27911, published online 15 June 2016
This Article contains errors.
For Fig. 6C, the lanes shown for JNK are incorrect. The correct Fig. 6 and accompanying legend appear below.
Effects of ACE2 and different treatments on the phosphorylation of MAPKs in lung tissue. LPS exposure caused a marked increase of the phosphorylation levels of p38 MAPK (A), ERK1/2 (B) and JNK (C), which was significantly suppressed by ACE2 overexpression in rat lung. Pretreatment with A779 or MLN-4760 completely abolished the inhibitory effects of ACE2 overexpression on LPS-induced p38 MAPK and ERK1/2 phosphorylation, but did not affect the level of JNK phosphorylation. ACE2 RNAi in rat lung significantly enhanced the LPS-induced ERK1/2 and JNK phosphorylation but did not change p38 MAPK phosphorylation. Data are represented as mean ± SD. *p < 0.05, versus control group; #p < 0.05, versus LPS group; $p < 0.05, versus ACE2 group (n = 6, per group).
In addition, full blots for the data presented were omitted in the original Article. They have been included in the attached Supplementary Information file.
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Li, Y., Zeng, Z., Cao, Y. et al. Author Correction: Angiotensin-converting enzyme 2 prevents lipopolysaccharide-induced rat acute lung injury via suppressing the ERK1/2 and NF-κB signaling pathways. Sci Rep 12, 5310 (2022). https://doi.org/10.1038/s41598-022-09404-5
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DOI: https://doi.org/10.1038/s41598-022-09404-5
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