Correction to: Scientific Reports https://doi.org/10.1038/s41598-017-15929-x, published online 22 November 2017

This Article contains an error in the order of the Figures. Figures 2, 3, 4 and 5 were published as Figures 4, 2, 5 and 3 respectively. The correct Figures 2, 3, 4 and 5 appear below. The Figure legends are correct.

Figure 2
figure 2

Examples of [1-13C]-acetate uptake in the aorta and kidneys over time. Hyperpolarized [1-13C]-acetate signal overlaid 1H-anatomical MR images of an axial slice, showing two kidneys and the presence of a signal in the aorta and following the kidneys. MR; magnetic resonance.

Figure 3
figure 3

13C-acetate in vivo hemodynamic parameters. Acetate perfusion (min/100 ml/mL), mean transit time (MTT) (sec), and acetate mean transit time metabolic clearance rate KMTT (min−1) before and after administration of furosemide. The mean is plotted with standard errors.

Figure 4
figure 4

Examples of 11C-acetate uptake in the aorta and kidneys over time. Positron emission tomography 11C-acetate signal overlaid 1H-anatomical MR images of a coronal slice, showing two kidneys and the presence of a signal in the aorta and following the kidneys. MR; magnetic resonance.

Figure 5
figure 5

11C-acetate in vivo kinetic parameters. (A) 11C-acetate single exponential metabolic clearance rate, Kmono. (B) 11C-acetate mean transit time metabolic clearance rate, KMTT. (C) Correlations between the decay derived or the first moment derived rates and the hyperpolarized 13C, showing a positive correlation (R2 = 0.82, P = 0.0003). (D) A tendency towards a similar response to furosemide treatment is seen between the 11C-PET and the 13C-hyperpolarization estimations. The mean is plotted with standard errors. PET, positron emission tomography.