Correction to: Nature Communications https://doi.org/10.1038/s41467-019-11845-y, published online 29 August 2019.
In the original version of this manuscript, in the discussion section in the seventh paragraph, the gene symbol for PARP14 was incorrectly given as PAR14 and incorrect citations of the literature were given. The incorrect version read ‘We would like to highlight variants in HLA-F, PAR14 and GAB2 controlled methylation sites in USP35. HLA-F is part of the nonclassical HLA-Ib genes, which are mono- or oligomorphic46. Surface expression of HLA-F has been demonstrated on activated T, B and NK cells, and serum IgG autoantibodies against HLA-F have been detected in SLE patients and correlated with disease activity63–65. PARP14 encodes for poly(ADP-ribose) polymerase (PARP) protein family 14 and is involved in cellular maintenance and cell fate decisions, such as cell-cycle progression, metabolic pathways and ribosome biogenesis66. Its role in SLE and autoimmune disease has not been defined but it has been shown to regulate glycolysis via IL-4 in B lymphocytes67 and to promote survival of cancer cells67–69.’
The correct version replaces these sentences with ‘We would like to highlight variants in HLA-F, PARP14 and GAB2 controlled methylation sites in USP35. HLA-F is part of the nonclassical HLA-Ib genes, which are mono- or oligomorphic46. Surface expression of HLA-F has been demonstrated on activated T, B and NK cells, and serum IgG autoantibodies against HLA-F have been detected in SLE patients and correlated with disease activity63–65. PARP14 encodes for poly(ADP-ribose) polymerase (PARP) protein family 14 and assists in post-translational ribosylation modification of target proteins. Its role in SLE and autoimmune disease has not been defined but it has been shown to regulate glycolysis via IL-4 in B lymphocytes66, promote survival of cancer cells67, and regulate macrophage activation68.’
Further, the original refs. 66–69 were replaced with the following corrected refs. 66–68 and all following references were renumbered.
All of these errors have now been corrected in the HTML and PDF versions of the article.
References
66. Cho, S. H. et al. Glycolytic rate and lymphomagenesis depend on PARP14, an ADP ribosyltransferase of the B aggressive lymphoma (BAL) family. Proc. Natl. Acad. Sci. USA 108, 15972–15977 (2011).
67. Iansante, V. et al. PARP14 promotes the Warburg effect in hepatocellular carcinoma by inhibiting JNK1-dependent PKM2 phosphorylation and activation. Nat. Commun. 6, 7882 (2015).
68. Iwata, H. et al. PARP9 and PARP14 cross-regulate macrophage activation via STAT1 ADP-ribosylation. Nat. Commun. 7, 12849 (2016).
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Lanata, C.M., Paranjpe, I., Nititham, J. et al. Author Correction: A phenotypic and genomics approach in a multi-ethnic cohort to subtype systemic lupus erythematosus. Nat Commun 11, 1164 (2020). https://doi.org/10.1038/s41467-020-15097-z
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DOI: https://doi.org/10.1038/s41467-020-15097-z
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