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Correction to: Cell Death and Disease (2016) 7, e2419; https://doi.org/10.1038/cddis.2016.268, Published online 13 October 2016.

Since the publication of the article, the authors became aware that Fig. 6a, d contained errors in the bands and loading controls. The newly compiled Fig. 6a, d is given below.

Fig. 6
Fig. 6

Knockdown of Mcl1 or Cdk4-induced cell cycle arrest and inhibited ES cell proliferation. a Mcl1 and Cdk4 siRNA analysis and expression check in murine and human ES cell lines resulted in upregulation of total RB and lower amount of pS780-RB. b Colony-forming assay in Matrigel displayed reduced clonogenic potency of EF-dependent cell lines after knockdown of Mcl1 or Cdk4 by siRNA treatment. c Cell cycle analysis of Mcl1 or Cdk4 siRNA treatment displayed enhanced G1 arrest. d In vitro growth inhibition of mouse and human EF-dependent cell lines by Palbociclib as well as the combinatorial treatment of Palbociclib and Obatoclax in triplicate. Cells were treated with inhibitors at the indicated concentration for 24 h. e CDK4/6 inhibition by Palbociclib displayed similar results as seen in the CDK4 siRNA assay

The authors apologise for any inconvenience caused.

Author information

Affiliations

  1. Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria

    • Tahereh Javaheri
    • ,  Jan Pencik
    • , Ha T. T. Pham
    • , Rahil Noorizadeh
    • , Barbara Sax
    • , Harini Nivarthi
    • , Michaela Schlederer
    • , Barbara Maurer
    • , Maximillian Hofbauer
    • , Marc Wiedner
    • , Lukas Kenner
    • , Florian Grebien
    •  & Richard Moriggl
  2. Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria

    • Tahereh Javaheri
    • , Ha T. T. Pham
    • , Rahil Noorizadeh
    • , Barbara Maurer
    •  & Richard Moriggl
  3. Medical University of Vienna, Vienna, Austria

    • Zahra Kazemi
    • ,  Jan Pencik
    • , Michaela Schlederer
    • , Lukas Kenner
    • , Mario Mikula
    • , Helmut Dolznig
    •  & Richard Moriggl
  4. Center of Physiology and Pharmacology, Vienna, Austria

    • Zahra Kazemi
  5. Clinical Institute of Pathology, Vienna, Austria

    • Jan Pencik
    • , Michaela Schlederer
    •  & Lukas Kenner
  6. Children’s Cancer Research Institute, St. Anna Kinderkrebsforschung, Vienna, Austria

    • Maximilian Kauer
    • , Dave N. T. Aryee
    • , Eleni M. Tomazou
    •  & Heinrich Kovar
  7. CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria

    • Harini Nivarthi
    •  & Marc Wiedner
  8. Department of Pediatrics, Medical University of Vienna, Vienna, Austria

    • Dave N. T. Aryee
    •  & Heinrich Kovar
  9. Randall Division of Cell and Molecular Biophysics, King’s College London, London, UK

    • Malcolm Logan
  10. Department of Bone and Skeletal Research, Institute of Experimental Musculoskeletal Medicine, University Hospital Münster, Münster, Germany

    • Christine Hartmann
  11. Institute of General Zoology and Endocrinology, University of Ulm, Ulm, Germany

    • Jan P. Tuckermann
  12. University of Veterinary Medicine, Vienna, Austria

    • Lukas Kenner
  13. Institute of Medical Genetics, Vienna, Austria

    • Mario Mikula
    •  & Helmut Dolznig
  14. Georgetown University Medical Center, Lombardi Comprehensive Cancer Center, Washington, USA

    • Aykut Üren
  15. Children’s Cancer Research Centre and Department of Pediatrics, Klinikum rechts der Isar, Technische Universität München, Munich, Germany

    • Günther H. Richter

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Correspondence to Richard Moriggl.

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DOI

https://doi.org/10.1038/s41419-018-0773-9