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Fig. 5 of this paper contains a mistake. The order of panels (b) and (c) should be reversed. For clarity, the entire figure is reprinted. We apologize for any inconvenience this may have caused.

Fig. 5 Engineered tetramers of Ang1 are useful in vivo reagents. a, Pharmacokinetic analysis of Ang-F1-Fc-F1 in mice, depicting circulating levels at indicated times after subcutaneous injection. b, In vivo phosphorylation of Tie2 stimulated by subcutaneous injection of Ang-F1-Fc-F1, assayed 24 h after injection. Tie2 was immunoprecipitated from 1 mg of lung lysate and immunoblotted either with anti-phosphotyrosine to detect phosphorylated Tie2 (pTie2) or with an antibody against Tie2 to detect total receptor. c, Systemic administration of Ang-F1-Fc-F1 protein causes resistance to mustard oil-induced vascular leakage comparable to resistance caused by adenoviral gene delivery. Plasma leakage of Evans blue was measured as described19 in mice treated with Ang-F1-Fc-F1 protein or with adenoviral vectors expressing either Ang1* (Ad Ang1*) or green fluorescent protein (Ad GFP) as a control. Data are expressed as mean ± SEM (standard error of the mean) for 3 ears per group (n = 3).