Loss of the transcription factor Gax is at the center of blood vessel dysregulation in the brain and contributes to Alzheimer disease pathology (pages 959–965).
This is a preview of subscription content, access via your institution
Access options
Subscribe to this journal
Receive 12 print issues and online access
$209.00 per year
only $17.42 per issue
Buy this article
- Purchase on Springer Link
- Instant access to full article PDF
Prices may be subject to local taxes which are calculated during checkout
Katie Ris
References
Iadecola, C. Nat. Rev. Neurosci. 5, 347–360 (2004).
Wu, Z. et al. Nat. Med. 11, 959–965 (2005).
Farkas, E. & Luiten, P.G. Prog. Neurobiol. 64, 575–611 (2001).
de la Torre, J.C. Stroke 33, 1152–1162 (2002).
Gorski, D.H. & Walsh, K. Trends Cardiovasc. Med. 13, 213–220 (2003).
Deane, R. et al. Neuron 43, 333–344 (2004).
Ward, N.L. & Lamanna, J.C. Neurol Res. 26, 870–883 (2004).
Park, L. et al. J. Neurosci. 25, 1769–1777 (2005).
Saito, T. et al. Regul. Pept. 127, 159–167 (2005).
Faraci, F.M. Stroke 36, 186–188 (2005).
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Iadecola, C. Rescuing troubled vessels in Alzheimer disease. Nat Med 11, 923–924 (2005). https://doi.org/10.1038/nm0905-923
Issue Date:
DOI: https://doi.org/10.1038/nm0905-923
This article is cited by
-
Cardiovascular drugs and triazole based kinase inhibitors as a new strategies for the treatment of Alzheimer disease
Russian Chemical Bulletin (2016)
