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Unlocking the pathogenesis of celiac disease

Nature Genetics volume 42, pages 281282 (2010) | Download Citation

A genome-wide association study reports more than a dozen new susceptibility loci for celiac disease. Analysis of eQTL data from these and previously established risk loci sheds light on the genetic pathways underlying this common autoimmune disease.

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Author information

Affiliations

  1. Robert M. Plenge is at the Division of Rheumatology, Immunology and Allergy, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.

    • Robert M Plenge

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Competing interests

The author declares no competing financial interests.

Corresponding author

Correspondence to Robert M Plenge.

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DOI

https://doi.org/10.1038/ng0410-281

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