Gubler, D.J. Dengue and dengue hemorrhagic fever. Clin. Microbiol. Rev. 11, 480–496 (1998).
Anders, K.L. et al. Epidemiological factors associated with dengue shock syndrome and mortality in hospitalized dengue patients in Ho Chi Minh City, Vietnam. Am. J. Trop. Med. Hyg. 84, 127–134 (2011).
Thai, K.T. et al. Seroprevalence of dengue antibodies, annual incidence and risk factors among children in southern Vietnam. Trop. Med. Int. Health 10, 379–386 (2005).
Loke, H. et al. Susceptibility to dengue hemorrhagic fever in vietnam: evidence of an association with variation in the vitamin D receptor and FC gamma receptor IIA genes. Am. J. Trop. Med. Hyg. 67, 102–106 (2002).
Loke, H. et al. Strong HLA class I–restricted T cell responses in dengue hemorrhagic fever: a double-edged sword? J. Infect. Dis. 184, 1369–1373 (2001).
Stephens, H.A. et al. HLA-A and -B allele associations with secondary dengue virus infections correlate with disease severity and the infecting viral serotype in ethnic Thais. Tissue Antigens 60, 309–318 (2002).
Sakuntabhai, A. et al. A variant in the CD209 promoter is associated with severity of dengue disease. Nat. Genet. 37, 507–513 (2005).
Vejbaesya, S. et al. TNF and LTA gene, allele and extended HLA haplotype associations with severe dengue virus infection in ethnic Thais. J. Infect. Dis. 199, 1442–1448 (2009).
Steinle, A. et al. Interactions of human NKG2D with its ligands MICA, MICB, and homologs of the mouse RAE-1 protein family. Immunogenetics 53, 279–287 (2001).
González, S., Lopez-Soto, A., Suarez-Alvarez, B., Lopez-Vazquez, A. & Lopez-Larrea, C. NKG2D ligands: key targets of the immune response. Trends Immunol. 29, 397–403 (2008).
Garrity, D., Call, M.E., Feng, J. & Wucherpfennig, K.W. The activating NKG2D receptor assembles in the membrane with two signaling dimers into a hexameric structure. Proc. Natl. Acad. Sci. USA 102, 7641–7646 (2005).
Hoang, L.T. et al. The early whole-blood transcriptional signature of dengue virus and features associated with progression to dengue shock syndrome in Vietnamese children and young adults. J. Virol. 84, 12982–12994 (2010).
Libraty, D.H. et al. Differing influences of virus burden and immune activation on disease severity in secondary dengue-3 virus infections. J. Infect. Dis. 185, 1213–1221 (2002).
Vaughn, D.W. et al. Dengue viremia titer, antibody response pattern, and virus serotype correlate with disease severity. J. Infect. Dis. 181, 2–9 (2000).
Kumar, V. et al. Genome-wide association study identifies a susceptibility locus for HCV-induced hepatocellular carcinoma. Nat. Genet. 43, 455–458 (2011).
Hinkes, B. et al. Positional cloning uncovers mutations in PLCE1 responsible for a nephrotic syndrome variant that may be reversible. Nat. Genet. 38, 1397–1405 (2006).
Chau, T.N. et al. Dengue virus infections and maternal antibody decay in a prospective birth cohort study of Vietnamese infants. J. Infect. Dis. 200, 1893–1900 (2009).
Tien, N.T. et al. A prospective cohort study of dengue infection in schoolchildren in Long Xuyen, Viet Nam. Trans. R. Soc. Trop. Med. Hyg. 104, 592–600 (2010).
Balmaseda, A. et al. Trends in patterns of dengue transmission over 4 years in a pediatric cohort study in Nicaragua. J. Infect. Dis. 201, 5–14 (2010).
Porter, K.R. et al. Epidemiology of dengue and dengue hemorrhagic fever in a cohort of adults living in Bandung, West Java, Indonesia. Am. J. Trop. Med. Hyg. 72, 60–66 (2005).
Endy, T.P. et al. Epidemiology of inapparent and symptomatic acute dengue virus infection: a prospective study of primary school children in Kamphaeng Phet, Thailand. Am. J. Epidemiol. 156, 40–51 (2002).
Mammen, M.P. et al. Spatial and temporal clustering of dengue virus transmission in Thai villages. PLoS Med. 5, e205 (2008).
Endy, T.P. et al. Determinants of inapparent and symptomatic dengue infection in a prospective study of primary school children in Kamphaeng Phet, Thailand. PLoS Negl. Trop. Dis. 5, e975 (2011).
Mells, G.F. et al. Genome-wide association study identifies 12 new susceptibility loci for primary biliary cirrhosis. Nat. Genet. 43, 329–332 (2011).
Purcell, S. et al. PLINK: a tool set for whole-genome association and population-based linkage analyses. Am. J. Hum. Genet. 81, 559–575 (2007).
Price, A.L. et al. Principal components analysis corrects for stratification in genome-wide association studies. Nat. Genet. 38, 904–909 (2006).
Purdue, M.P. et al. Genome-wide association study of renal cell carcinoma identifies two susceptibility loci on 2p21 and 11q13.3. Nat. Genet. 43, 60–65 (2011).
Thomas, G. et al. A multistage genome-wide association study in breast cancer identifies two new risk alleles at 1p11.2 and 14q24.1 (RAD51L1). Nat. Genet. 41, 579–584 (2009).
Lettre, G., Lange, C. & Hirschhorn, J.N. Genetic model testing and statistical power in population-based association studies of quantitative traits. Genet. Epidemiol. 31, 358–362 (2007).
McGovern, D.P. et al. Genome-wide association identifies multiple ulcerative colitis susceptibility loci. Nat. Genet. 42, 332–337 (2010).
Asano, K. et al. A genome-wide association study identifies three new susceptibility loci for ulcerative colitis in the Japanese population. Nat. Genet. 41, 1325–1329 (2009).
Barrett, J.C., Fry, B., Maller, J. & Daly, M.J. Haploview: analysis and visualization of LD and haplotype maps. Bioinformatics 21, 263–265 (2005).