Abstract
TGF-β is a cytokine that enhances wound healing, a process involving diverse TGF-β-responsive cell types. Mice with a deletion of the Smad3 gene exhibit an increased rate of wound healing. The lack of Smad3 seems to modify the cellular response to TGF-β and may suggest an intriguing molecular basis to this process.
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Massagué, J. Wounding Smad. Nat Cell Biol 1, E117–E119 (1999). https://doi.org/10.1038/12944
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DOI: https://doi.org/10.1038/12944
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