CXCL12 promotes glycolytic reprogramming in acute myeloid leukemia cells via the CXCR4/mTOR axis

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MQ was supported by the IZKF Erlangen. RJ was supported by the ELAN program of the University of Erlangen. FAK was supported by the EC FP7-funded project RADIANT. DM was supported by the IZKF Erlangen and the Max-Eder program of the Deutsche Krebshilfe. We acknowledge the assistance of the Core Unit Cell Sorting and Immunomonitoring Erlangen.

Author contributions

MB performed research, analyzed data and helped writing the manuscript. MQ, MB, FK, DS and WH performed research and analyzed data. MA collected patient material and provided patient data. AM provided patient material and helped writing the manuscript. RJ performed research, helped designing the study and writing the manuscript. DM designed the study, analyzed data and wrote the manuscript.

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Correspondence to D Mougiakakos.

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The authors declare no conflict of interest.

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Braun, M., Qorraj, M., Büttner, M. et al. CXCL12 promotes glycolytic reprogramming in acute myeloid leukemia cells via the CXCR4/mTOR axis. Leukemia 30, 1788–1792 (2016).

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