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The anti-apoptotic gene BCL2A1 is a novel transcriptional target of PU.1

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We thank Deborah Shan, Judith Laedrach, and Gustav Arvidsson for expert technical support. This work was supported by grants from the Swiss National Science Foundation 3100A0-118276 (to MPT) and 3100A0-112385 (to MFF and AT), the Bern University Research Foundation (to MPT), the Werner and Hedy Berger-Janser Foundation of Cancer Research (to MFF and MPT), the NIH, HL091219 and DK54938, the Joyce Klein Stock Gift (to BET), the Marlies-Schwegler Foundation, the Ursula-Hecht-Foundation for Leukemia Research, and the Bernese Foundation of Cancer Research (to MFF).

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Correspondence to M P Tschan.

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MJ designed the project, performed experiments, collected and analyzed all data, manuscript writing; JB was involved in knockdown experiments and expression analysis; VAR performed microarray analysis; TH provided AML samples, expression analysis; AT, MFF, and BET performed project planning and design, reviewed manuscript; MPT was the principal investigator and takes primary responsibility for the paper.

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Jenal, M., Batliner, J., Reddy, V. et al. The anti-apoptotic gene BCL2A1 is a novel transcriptional target of PU.1. Leukemia 24, 1073–1076 (2010).

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