How great is the risk of future bleeding for children with brain arteriovenous malformations?
Louis R Caplan
Correspondence Beth Israel Deaconess Medical Center, Department of Neurology, Division of Cerebrovascular Disease, 330 Brookline Avenue, Palmer 132, Boston, MA 02215, USA
Email lcaplan@caregroup.harvard.edu
This article has no abstract so we have provided the first paragraph of the full text.
AVMs are characterized by direct connections between arteries and veins without any capillaries in between. AVMs are heterogeneous—their complexity varies from single shunts between one artery and one vein (arteriovenous fistulas) to intricate conglomerates with multiple feeding arteries and many draining veins communicating through multiple shunts.1 Histologically, AVMs are composed of feeding arteries, draining veins and an angiomatous nidus. Tortuous and dilated vessels within the nidus are indistinguishable as arteries or veins, but nothing within the lesion resembles a true capillary bed.
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