NMDA receptors are present on oligodendrocyte processes and are activated during ischemia
Christine Kyme
This article has no abstract so we have provided the first paragraph of the full text.
Oligodendrocytes are responsible for axon myelination within the CNS. Glutamate-mediated damage to these cells results in impaired axonal saltatory conduction, and contributes to diseases such as multiple sclerosis, cerebral palsy and ischemia. Three papers recently published in Nature report new findings that oligodendrocyte damage occurs not only via glutamatergic 
-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)/kainate receptors, but also via N-methyl-D-aspartate (NMDA) receptors, which were previously not thought to be present on this cell type.
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