L-arginine therapy following myocardial infarction is ineffective and possibly unsafe
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Nitric oxide deficiency is associated with endothelial dysfunction. L-arginine, a substrate for nitric oxide synthase, has been shown to improve vascular endothelial function. The VINTAGE MI trial attempted to assess the effect of L-arginine therapy on vascular stiffness and left ventricular function following a first ST-segment elevation myocardial infarction.
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