Selected examples of gene-environment interactions observed in at least two studies
Selected examples of gene-environment interactions observed in at least two studies
ADH1C, alcohol dehydrogenase 1C (class I), γ-polypeptide; ADRB2, adrenergic, β-2-, receptor, surface; CCR5, chemokine (C–C motif) receptor 5; APOE, apolipoprotein E; F5, coagulation factor V; HIV, human immunodeficiency virus; HLA-DPB1, major histocompatibility complex, class II, DP β-1; MC1R, melanocortin receptor 1; MTHFR, 5,10-methylenetetrahydrofolate reductase (NADPH); NAT2, N-acetyltransferase 2; PPARG2, peroxisome proliferative activated receptor-γ; TPMT, thiopurine
S-methyltransferase; UGT1A6, UDP glycosyltransferase 1 family, polypeptide A6.
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Genetics seemed to promise incredible advances in the fight against disease, yet new cures and treatments have been slow to arrive. Why?
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