The author is in the Department of Molecular Biophysics and Biochemistry, Yale University School of Medicine, Sterling Hall of Medicine, 333 Cedar Street, New Haven, Connecticut 06520, USA. patrick.sung@yale.edu
The tumor suppressor protein BRCA2 promotes efficient repair of damaged chromosomes by homologous recombination, and it does so by influencing the activity of the Rad51 recombinase. A new biochemical study on the Ustilago maydis BRCA2 ortholog Brh2 sheds light on the molecular function of this tumor suppressor in the Rad51-mediated homologous recombination reaction.
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