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Nature Reviews Cancer 9, 457 (1 July 2009) | doi:10.1038/nrc2689

Therapeutics: Dependent on KRAS

Sarah Seton-Rogers

Although activating KRAS mutations occur frequently in cancer, targeting this oncogene has proved difficult. Three groups have now identified pathways that have not been previously linked to KRAS — all of which may provide pharmacologically tractable anticancer targets — and that are crucial in cancer cells dependent on activated KRAS.