Brief Communication abstract


Nature Methods 5, 951 - 953 (2008)
Published online: 19 October 2008 | doi:10.1038/nmeth.1261

Linking SNPs to CAG repeat length in Huntington's disease patients

Wanzhao Liu1, Lori A Kennington1, H Diana Rosas2, Steven Hersch2, Jang-Ho Cha2, Phillip D Zamore3 & Neil Aronin1

Top

Allele-specific silencing using small interfering RNAs targeting heterozygous single-nucleotide polymorphisms (SNPs) is a promising therapy for human trinucleotide repeat diseases such as Huntington's disease. Linking SNP identities to the two HTT alleles, normal and disease-causing, is a prerequisite for allele-specific RNA interference. Here we describe a method, SNP linkage by circularization (SLiC), to identify linkage between CAG repeat length and nucleotide identity of heterozygous SNPs using Huntington's disease patient peripheral blood samples.

Top
  1. Department of Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA.
  2. MassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital, 16th Street, Charlestown, Massachusetts 02129, USA.
  3. Department of Biochemistry and Molecular Pharmacology, and Howard Hughes Medical Institute, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA.

Correspondence to: Phillip D Zamore3 e-mail: phillip.zamore@umassmed.edu

Correspondence to: Neil Aronin1 e-mail: neil.aronin@umassmed.edu



MORE ARTICLES LIKE THIS

These links to content published by NPG are automatically generated.

NEWS AND VIEWS

Expanded CAG repeats in the crosshairs

Nature Biotechnology News and Views (01 May 2009)

Huntingtin's critical cleavage

Nature Neuroscience News and Views (01 Sep 2006)


Extra navigation

Apply for your free subscription to
Nature Methods

Subscribe

Open Innovation Challenges

naturejobs

ADVERTISEMENT