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Nature Medicine  9, 1363 - 1369 (2003)
Published online: 12 October 2003; | doi:10.1038/nm947

Hepatocyte growth factor and its receptor are required for malaria infection

Margarida Carrolo, Silvia Giordano, Laura Cabrita-Santos, Simona Corso, Ana M Vigário, Susana Silva, Patricia Leirião, Daniel Carapau, Rosario Armas-Portela, Paolo M Comoglio, Ana Rodriguez & Maria M Mota
 
Figure 1 thumbnailFigure 1.
Sporozoite migration through cells and mechanical cell wounding induce the release of ISIF.

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HGF secreted by sporozoite-traversed host cells is required for infection.

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Figure 3 thumbnailFigure 3.
HGF activity correlates with infection level.

Full FigureFull Figure and legend (19K)		 Figure 4 thumbnailFigure 4.
Effect of HGF on Plasmodium infection is mediated through its receptor, MET.

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Figure 5 thumbnailFigure 5.
HGF/MET signaling is required for early development of parasites inside host cells, and involves host-cell actin cytoskeleton reorganization.

Full FigureFull Figure and legend (52K)		 Figure 6 thumbnailFigure 6.
MET activation is required during natural in vivo malarial infection.

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