Remembering MOG: Autoantibody mediated demyelination in multiple sclerosis?
Hartmut Wekerle
Max−Planck Institute for Neurobiology
D−82152 Martinsried Germany
A comparative study of acute lesions in multiple sclerosis patients and
in marmosets suggests that anti−MOG antibodies are responsible for the
demyelination characteristic of this autoimmune disease (170−175).