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Letter
Nature Medicine 15, 788-793 (1 July 2009) | doi:10.1038/nm.1980;
Activation of kinin receptor B1 limits encephalitogenic T lymphocyte recruitment to the central nervous system
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Abstract
Previous proteomic and transcriptional analyses of multiple sclerosis lesions revealed modulation of the renin-angiotensin and the opposing kallikrein-kinin pathways. Here we identify kinin receptor B1 (Bdkrb1) as a specific modulator of immune cell entry into the central nervous system (CNS).
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