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NAMPT is essential for the G-CSF–induced myeloid differentiation via a NAD+–sirtuin-1–dependent pathway

Julia Skokowa, Dan Lan, Basant Kumar Thakur, Fei Wang, Kshama Gupta, Gunnar Cario, Annette Müller Brechlin, Axel Schambach, Lars Hinrichsen, Gustav Meyer, Matthias Gaestel, Martin Stanulla, Qiang Tong & Karl Welte

Nature Medicine 15, 151 - 158 (2009) Published online: 1 February 2009

doi:10.1038/nm.1913

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

G-CSF induces NAMPT and NAD+ in CD33+ myeloid progenitors from healthy individuals and individuals with congenital neutropenia in vivo and in vitro.

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Figure 2

NAMPT triggers myeloid differentiation of CD34+ cells from healthy individuals.

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Figure 3

Nicotinamide induces in vitro myeloid differentiation of CD34+ cells from healthy individuals.

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Figure 4

NAMPT is capable of G-CSF–dependent granulocytic differentiation.

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Figure 5

SIRT1 is involved in NAMPT-C/EBP–dependent activation of G-CSF and G-CSFR.

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Figure 6

Treatment of healthy individuals with vitamin B3 leads to increased numbers of neutrophils in peripheral blood.

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