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Neurotensin increases mortality and mast cells reduce neurotensin levels in a mouse model of sepsis

Adrian M Piliponsky, Ching-Cheng Chen, Toshihiko Nishimura, Martin Metz, Eon J Rios, Paul R Dobner, Etsuko Wada, Keiji Wada, Sherma Zacharias, Uma M Mohanasundaram, James D Faix, Magnus Abrink, Gunnar Pejler, Ronald G Pearl, Mindy Tsai & Stephen J Galli

Nature Medicine 14, 392 - 398 (2008) Published online: 30 March 2008

doi:10.1038/nm1738

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

NT levels are increased and contribute to mortality after CLP in mice.

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Figure 2

Evidence that NT can contribute to mortality after CLP in wild-type or KitW/W-v MC-deficient mice.

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Figure 3

MCs reduce peritoneal NT concentrations and NT-induced hypotension.

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Figure 4

NLN contributes to the ability of MCs to reduce amount of NT and NT-induced hypotension.

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Figure 5

MCs can degrade NT in the absence of extensive degranulation.

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Figure 6

MC expression of Ntsr1 is required for optimal MC-dependent reduction of NT-induced hypotension and enhancement of survival after CLP.

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