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From the following article

Transcription factor FOXO3a controls the persistence of memory CD4+ T cells during HIV infection

Julien van Grevenynghe, Francesco A Procopio, Zhong He, Nicolas Chomont, Catherine Riou, Yuwei Zhang, Sylvain Gimmig, Genevieve Boucher, Peter Wilkinson, Yu Shi, Bader Yassine-Diab, Elias A Said, Lydie Trautmann, Mohamed El Far, Robert S Balderas, Mohamed-Rachid Boulassel, Jean-Pierre Routy, Elias K Haddad & Rafick-Pierre Sekaly

Nature Medicine 14, 266 - 274 (2008) Published online: 2 March 2008

doi:10.1038/nm1728

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

Memory TCM and TEM cells from EC subjects are less sensitive to Fas-mediated apoptosis and show heightened in vitro persistence after multiple rounds of TCR stimulation compared to those from ST subjects or HIV- donors.

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Figure 2

mDCs from EC subjects, ST subjects and HIV- subjects show similar phenotypic and functional patterns.

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Figure 3

Ex vivo TCM and TEM cells from EC and ST subjects show similar profiles of activation and proliferation markers.

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Figure 4

TCM and TEM from EC subjects display higher phospho-FOXO3a phenotypes than those from ST subjects and HIV- subjects.

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Figure 5

FOXO3a shutdown by siRNA rescues TCM and TEM cells from ST donors from Fas-mediated apoptosis.

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Figure 6

FOXO3a Nt increases TCM cell persistence of ST subjects.

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