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Nature Medicine 13, 403 - 404 (2007)
doi:10.1038/nm0407-403
Netting bacteria in sepsis
Constantin Urban1 & Arturo Zychlinsky1
- The authors are in the Department of Cellular Microbiology, Max Planck Institute for Infection Biology, Charitéplatz 1, Berlin 10117, Germany. e-mail: zychlinsky@mpiib-berlin.mpg.de
Abstract
Platelets act as intermediaries in the pathogenesis of sepsis—sensing bacteria and signaling neutrophils to release fibrous traps that remove bacteria from the bloodstream. This response may also contribute to tissue injury (pages 463–469).
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